Hello Bluebird.
I hope we can help each other with our combined research. Main difference I can see is that neither my mother nor I have it on our hands and the history as below.
I have posted in a two forums about the rash that both my mother and I developed about 6 weeks ago.
My case study-
My initial hypothetical –
1. shedding of Covid vax nano particles/spike as studied extensively by Dr P. Kory. (Neither my mother or myself have had Covid, nor been injected with it. I did have a bad gastic flu last year but tested negative for Covid. Four days ago I had blood drawn for Covid antibodies and the nucleocapsid privately as no Dr is likely to order them through Medicare) as per discussed here if positive to nuclecapsid then infection with virus, if not, only Covid antibodies then injection (my addition ??shedding) – https://jpsaleebymd.substack.com/p/neuropasc-aka-neurological-covid?utm_source=substack&utm_medium=email#media-6a6bf92d-f838-45cb-82a2-a11d4c6041e3
2. Bed bugs – suggested by another member on this Forum, not found, all bedding washed etc.
3. Rainwater – filter stopped way ahead of time, many pollutants in air that land on roof, rainwater tanks getting low as no rain all summer except one night (?January but possibly not more than would have been flushed away in first flush), red dust toxin report from Alcoa mine and refinery 20 kms to the east (prevailing summer wind is from the east). Awaiting water test & hair test report.
My 94 year old mother does not suffer allergies or ill health other than dementia (not Alzheimer’s), not on medication etc whereas I suffered over 9 months of what was possibly idiopathic chronic urticaria in September 2020. No cause found, no systemic inflammation etc and biopsy – spongiosis. (I am now having other thoughts about its origin although Covid was rare in Western Australia – highly quarantined state, and I was unlikely to have come in contact with it then & pre jabs.)
We had an overseas guest stay for three weeks in February this year. One week after their departure (approximately 4 weeks after starting to drink rainwater without filtering it) both my mother and I developed an itchy rash on chest and back and then upper legs. The rash has subsided significantly in my mother but has continued in myself. I used betamethasone cream liberally for my mother, whereas I have used a mixture of fresh aloe vera plant, healing oils, and oatmeal like anti itch creams. Systemic antihistamines were tried a few times with no lasting effect (a couple of hours maybe). My mother still has some marks on her back and scratches occasionally but is not disturbed by the rash. Up until yesterday I was going mad with the itching (varies day to day and area).
I was already taking Vit D, Quecertin, & Resveratrol more recently, and a formula for varicose veins so ordered NAC, Nattokinase, Serrapeptase. I have also ordered Ivermectin from India (hope it gets through customs). See photo – ones to be taken on empty stomach are first.
I started taking the new supplements on the 30th (now taken 10 doses) and have felt no different – tired but able to push through and do gardening, cleaning etc for a 2-3 hours per day. I have used betamethasone cream a couple of times when itch unbearable including when my left lower leg was swollen and oozing (venous insufficiency). I also started wearing support stockings again which has helped. The connection with venous insufficiency is also supported by this study information on id reactions and autoeczematisation. Note the connection to T lymphocytes, proinflammatory cytokines and the link to viruses.
Autoeczematisation
“Approximately 37% of patients diagnosed with stasis dermatitis develop an id reaction (Figure 1).
The pathogenesis of AE is uncertain, but circulating T lymphocytes play a role in this reaction. Normally, T cells are activated by a release of antigens after a primary exposure to a stimulus. However, overactivation of these T cells induces autoimmune reactions such as AE.7 Activated T lymphocytes express HLA-DR and IL-2 receptor, markers elevated in the peripheral blood of patients undergoing id reactions. After treatment, the levels of activated T lymphocytes decline. An increase in the number of CD25+ T cells and a decrease in the number of suppressor T cells in the blood may occur during an id reaction.7-9 Keratinocytes produce proinflammatory cytokines, such as thymic stromal erythropoietin, IL-25, and IL-33, that activate T cells.10-12 Therefore, the most likely pathogenesis of an id reaction is that T lymphocytes are activated at the primary reaction site due to proinflammatory cytokines released by keratinocytes. These activated T cells then travel systemically via hematogenous dissemination.
The spread of activated T lymphocytes produces an eczematous reaction at secondary locations distant to the primary site.9
…
In later stages, vesicles disseminate to the legs, arms, and trunk, where they group to form papules and nummular patches in a symmetrical pattern.5,13-15 These lesions may be extremely pruritic. The pruritus may be so intense that it interrupts daily activities and disrupts the ability to fall or stay asleep.16
…
Viral infections that can cause an id reaction are herpes simplex virus and molluscum contagiosum.27-29 Scabies, leishmaniasis, and pediculosis capitis are parasitic infections that may be etiologic.14,30,31 In addition, noninfectious stimuli besides stasis dermatitis that can produce id reactions…”
https://www.mdedge.com/dermatology/article/245813/contact-dermatitis/autoeczematization-strange-id-reaction-skin
You may find this 2021 research helpful if getting a biopsy –
Covid and eczema
https://www.ncbi.nlm.nih.gov/pmc/articles/PMC8431833/
Here are some photos of the rash on my mother (26th February – early onset & myself on the 2nd March although I had it on the 26th as well. I think it started a few days earlier on the chest). This is my left leg last night when the itch got so bad I used betamethasone cream mainly on the right leg and today both are reduced in itch and redness especially the right leg.
References on FLCCC site
Fibrin – Nattokinase can break down fibrin to potentially prevent clots, reduce BP, blood thinning, ?amyloid fibrils, Take 2000 to 4000 FU twice a day
https://imahealth.org/wp-content/uploads/2023/11/Dr.-Carmans-Guide-to-Nattokinase-.pdf
Shedding is real
https://imahealth.org/wp-content/uploads/2024/02/conf2024-Shedding%20is%20Real-kory-slides.pdf
Skin rashes one of most common symptoms