Jeff Gerber
Forum Replies Created
-
I’ve been taking the 3x a day (600mg) of Augmented NAC since May 7th. I’ve gone beyond the 3 months and with no side effects. My wife did it with me, but she ended at the 3-month mark. I think she only noticed subtle GI issues like loose stool at the very beginning, but the body adjusted.
You wouldn’t happen to be taking Ivermectin as well, would you? I know that too high of a dose of Ivermectin can be associated with blurred vision, in which case you simply lower the dose.
I’m trying to stretch my imagination on NAC and vision but not coming up with how the two could be connected.
-
See this article:
“Here we refer to it as Long COVID/PASC. Lingering symptoms persist for as much as 6 months (or longer) after acute infection, where COVID-19 survivors complain of recurring fatigue or muscle weakness, being out of breath, sleep difficulties, and anxiety or depression. Given that blood clots can block microcapillaries and thereby inhibit oxygen exchange, we here investigate if the lingering symptoms that individuals with Long COVID/PASC manifest might be due to the presence of persistent circulating plasma microclots that are resistant to fibrinolysis.“
cardiab.biomedcentral.com
Background Severe acute respiratory syndrome coronavirus 2 (SARS-Cov-2)-induced infection, the cause of coronavirus disease 2019 (COVID-19), is characterized by acute clinical pathologies, including various coagulopathies that may be accompanied by hypercoagulation and platelet hyperactivation. Recently, a new COVID-19 phenotype has … Continue reading
-
I asked Copilot the following question: “If a microclot is resistant to fibrinolysis would this cause a d-dimer test to be ineffective at identification?”
Copilot answer:
Yes — that’s exactly the kind of scenario where a D‑dimer test can give a misleadingly low result.
Here’s why:
– **What D‑dimer measures** — It detects fragments of cross‑linked fibrin that are released when a clot is broken down by the body’s fibrinolytic system.
– **The catch with fibrinolysis‑resistant microclots** — Some microclots, especially those with abnormal fibrin structures (like fibrin‑amyloid or “fibrinaloid” forms), are unusually resistant to plasmin‑mediated breakdown. If they aren’t being degraded, they won’t release much D‑dimer into circulation.
– **Observed pattern** — Studies have found cases where patients had a high burden of microclots but low D‑dimer levels, because impaired plasmin generation or altered fibrin architecture prevented normal lysis.
– **Implication** — A normal or low D‑dimer result doesn’t necessarily rule out the presence of such persistent microclots. Detecting them may require specialized assays, such as fluorescence microscopy of platelet‑poor plasma stained with amyloid‑binding dyes like Thioflavin T.
In short, if the clot isn’t breaking down, the D‑dimer “signal” stays quiet — even if the microclots themselves are still causing trouble.
-
💯
I think of it the same as heavy metals. It doesn’t matter where it comes from, the sum total that you have in your body is what matters. Just like heavy metals, it’s very difficult to get rid of. What stuck in my head recently is watching a YouTube video on the simple topic of micro clots. Part 1 of 3 – What Are Microclots in Long COVID?
In that video (see attached screenshot) they show how the fibrin (the fibers that make up a clot) look completely different if they have spike protein involved. The body knows how to break down fibrin, that’s how it does things normally, but not fibrin with spike protein influencing it. The fibrin becomes very mutated in appearance like knotted rope. Now imagine this crap (technical term 🙄) all over the body, messing with all different systems. It becomes pretty obvious (at least to me) that it has a cumulative effect the more you are exposed. In that video series she even talks about the tests they ran and how difficult it was to break it down.
I think it’s brilliant they are pointing this out because, just maybe, it will help people come to the conclusion that no one should ever desire to be injected with spike protein.
-
The link above is broken because it contained extra characters. Here it is.
Hazan: Cholinergic eubiosis: A hypothesis on Ivermectin-upregulated Bifidobacterium
c19early.org
Hazan: Cholinergic eubiosis: A hypothesis on Ivermectin-upregulated Bifidobacterium
Hypothesis that ivermectin may increase beneficial Bifidobacterium populations in the gut through a "cholinergic eubiosis" mechanism. Authors..
-
Here’s another interesting twist and it leads to some interesting ideas for what else a person might try when they get sick with Norovirus or Rotavirus. It might be beneficial to consume things that lower gut pH and increase short chain fatty acids.
I asked, “Does viral gastroenteritis raise the pH of the gut?”
Copilot:
The short answer is: it can, but not always — and the effect is usually indirect.
Viral gastroenteritis (from norovirus, rotavirus, etc.) disrupts the gut environment in several ways that can shift pH, but the direction of change depends on what’s happening in the microbiome and with digestion.
🔬 Mechanisms That Can Raise Gut pH
- Loss of acid-producing bacteria – Beneficial microbes like Bifidobacterium and Lactobacillus produce lactic acid and short-chain fatty acids (SCFAs) that keep the colon slightly acidic (pH ~5.5–6.5). Vomiting and diarrhea can flush these microbes out, reducing acid production and allowing pH to drift upward toward neutral.
- Reduced fermentation – If the infection damages the small intestine’s absorptive surface, undigested carbohydrates may not reach the colon in the same way, lowering SCFA output and raising pH.
- Bile acid malabsorption – Some viral infections temporarily impair bile reabsorption, and excess bile salts in the colon can increase luminal pH.
⚖️ Mechanisms That Can Lower Gut pH
- Malabsorption of carbohydrates – If sugars reach the colon in larger amounts, surviving bacteria may ferment them rapidly, producing more acids and lowering pH.
- Inflammatory response – Certain immune-mediated changes can favor acid-producing bacterial blooms in the short term.
📚 What Studies Suggest
- In rotavirus infections, animal and pediatric studies have shown a temporary rise in fecal pH during acute illness, often linked to loss of bifidobacteria and lactobacilli.
- In norovirus, data are more limited, but similar microbiome disruptions have been observed, suggesting pH could shift upward during or after infection.
- These pH changes are usually transient — as the microbiome recovers, SCFA production resumes and pH moves back toward its normal slightly acidic range.
🧠 Why This Matters
A higher gut pH during or after viral gastroenteritis can:
- Make the colon more hospitable to opportunistic pathogens
- Slow recovery of beneficial bacteria
- Alter immune signaling in the gut
This is one reason why restoring acid-producing microbes (through diet, prebiotics, or targeted probiotics) is often recommended after severe gastroenteritis.
Lowering gut pH and boosting short-chain fatty acids (SCFAs) like butyrate, acetate, and propionate go hand in hand — because SCFAs themselves are acidic, and their production naturally shifts the colon toward a healthier, slightly acidic range (pH ~5.5–6.5).
Here’s a science-backed playbook for achieving both goals:
🥦 1. Feed the Right Microbes With Fermentable Fibers
- Resistant starches – e.g., cooked-and-cooled potatoes, green bananas, legumes, rice that’s been chilled. These are prime substrates for butyrate-producing bacteria.
- Soluble fibers – from oats (beta-glucan), psyllium husk, chia seeds, flaxseeds, and pectins in apples/citrus.
- Inulin & fructooligosaccharides (FOS) – found in onions, garlic, leeks, asparagus, Jerusalem artichokes. These selectively feed Bifidobacterium and Lactobacillus, which lower pH via lactic acid and SCFA production.
🥛 2. Introduce Acid-Producing Probiotics
- Bifidobacterium and Lactobacillus strains produce lactic acid, which can be cross-fed to butyrate producers like Faecalibacterium prausnitzii.
- Fermented foods (kefir, yogurt, sauerkraut, kimchi) can help reseed these populations after disruption.
🍠 3. Use Polyphenol-Rich Plant Foods
- Berries, cocoa, green tea, and colorful vegetables contain polyphenols that beneficial bacteria metabolize into acids, lowering pH and supporting SCFA producers.
- Polyphenols can also suppress pH-raising, proteolytic bacteria.
🥣 4. Moderate Protein & Emphasize Plant-Based Meals
- Excess protein fermentation tends to raise pH (producing ammonia and amines).
- Balancing protein with high-fiber plant foods shifts fermentation toward carbohydrate-derived SCFAs.
🧪 5. Consider Direct SCFA or Prebiotic Supplementation
- Butyrate salts (e.g., sodium butyrate) can directly acidify the colon and feed colonocytes.
- Prebiotic blends (resistant starch + inulin) have been shown to synergistically raise SCFA output and lower pH.
🚰 6. Support Microbial Cross-Feeding
- Pair lactic acid–producing bacteria with fiber that butyrate producers can use.
- Example: yogurt (lactate source) + oats (beta-glucan) → lactate converted to butyrate by secondary fermenters.
-
I’m not a medical professional, but as a person who is dealing with post-vaccine syndrome, I strongly suggest you do more than ivermectin and look at what IMA-HelenT pointed to. For example:
Nattokinase: This highly effective fibrinolytic and antiplatelet agent targets the abnormal clotting that can occur from spike protein-related disease.
When I read heart issues, this one pops to mind especially because it helps to address blood clots. It’s OTC, but, you do want to have a physician give the go ahead. If you were on blood thinners etc. it can be contraindicated. https://youtu.be/22HJmgzrpqw will give you an idea of just how difficult it is to treat micro clots and why they occur. But this is only one aspect. Spike is very difficult to clean up, but when you do start cleaning it up you can have some fantastic turn around in your symptoms.
The protocol has a LOT of bright doctors and thinking that went into it. Ivermectin is good stuff, but it is only one angle on a complex problem.
-
There is also a very close connection between the IMA protocol and Dr. Peter MCCullough’s:
Dr. Peter A. McCullough, MD, MPH
He most recently added to his overall protocol:
The gout medicine Dr. Peter McCullough has mentioned in his expanded protocol is **colchicine**.
In his “Base Spike Detox” framework, colchicine isn’t part of the core triple combination (nattokinase, bromelain, curcumin) — instead, he lists it as an *additional treatment* when there are **significant pleuropericardial problems** (inflammation involving the membranes around the heart and lungs).
Personally, the threat of blood clots is very real to me in having post vaccine syndrome myself. Further, my brother died a very unusual and rare death back in May where he had a massive bilateral thrombosis (in both legs at the same time). Yes, he was vaccinated. No, I didn’t understand the risk and protocol in time to save him.
The problem is:
1) People don’t know they have micro clots forming – as stated in the video above, you need someone with a platelet aggregometer (not a common clinical item) and the blood needs to be processed locally immediately after being drawn and can’t be sent to a lab due to the platelet instability.
2) The Hybrid Hypothesis Study here in IMAHealth shows the plausibility of how you can continue to build and contribute to problems in the body the more you are exposed to spike protein either from viral infection or injection. i.e. it can sneak up on you.
3) If you do identify that you have a problem with micro clots, the spike makes it VERY difficult to break it down.
4) Outside of these protocols mentioned, as far as I know, there is no good way to address these clots. It’s the best we’ve got.
-
I-PREVENT: Vaccine Injury – Independent Medical Alliance is the protocol.
“The primary approach to preventing delayed complications from vaccination is to enhance the body’s ability to eliminate spike protein. This is best achieved by practicing intermittent fasting/time-restricted eating and with a supplement like resveratrol, which activates autophagy and encourages the removal of spike protein. In addition, nattokinase, a naturally derived enzyme, breaks down extracellular spike protein and is a potent fibrinolytic agent, which breaks down blood clots. Furthermore, treating hyperinsulinemia likely limits both endothelial inflammation and carcinogenesis. We have added other interventions to this core treatment approach that likely have additional benefits. These include anti-platelet and fibrinolytic agents, which are central to the prevention of cardiovascular events following vaccination.”
-
I would have to retrace my steps (forgive me, it’s late and I’ve had a long day), but I believe the answer is yes, spike protein can be transmitted in semen. It is a known fact that it has been found in the testes.
-
I wholeheartedly agree with you. Our current understanding of health and sickness as a society is hyper focused on two false premises:
1. Packaged and processed foods exist which are good for you
2. The way to fix disease and sickness is always with medication or surgery
The third element that we don’t see because of the modern society we’ve grown up in is that we believe we will “starve to death” if we go a period without food. There’s also a notion of “I won’t get protein if I don’t eat red meat”.
Dr. Patrick Soon-Shiong, MD and Dr. Valter Longo, PhD both have looked beyond these premises and looked at our design and biology. Dr. Soon-Shiong mentions natural killer (NK) cells have existed for 450 million years (Cambrian era when the diversity of life exploded) and these are a foundation upon which to build medical science… something we have still not embraced and is the reason why his approach works. Dr. Valter Longo also relies on an ancient biological design which is that we have survived periods of starvation for millions of years and this system can be used by fasting in order to put the body into a protective mode and do some house cleaning.
If we’re not using either of these approaches as our first line of defense, it’s like trying to put out a fire with a squirt gun. It might do a bit of something, but it isn’t addressing the issue at large.
-
I forgot to mention I have been doing red light therapy at home with a panel array and this allows me to also target the thyroid fairly directly.
-
Hi @surf302 ,
Yes, I have done a follow up test. Here’s a brief history:
6/30/2023: Pfizer bivalent vaccination
3/4/2024: SARS-CoV-2 Spike Ab Dilution = >25,000
3/27/2025: SARS-CoV-2 Spike Ab Dilution = 10177
5/1/2025: Started Ivermectin + Augmented NAC
5/4/2025: Started low dose nicotine patches
5/7/2025: Felt far better than I could remember, increase in energy, POTS symptoms went away, taste and smell improved, mental clarity…
6/30/2025: SARS-CoV-2 Spike Ab Dilution = 10487
The number is essentially the same. The body remembers antigens like spike protein.
In my case, I am vaccine injured (post vaccine syndrome) and unfortunately, I wasn’t wise to the issue until I had been exposed to all 3 vaccines (Janssen, Pfizer, Moderna). Dr. Saleeby states that the worst cases he has seen have been exactly this, people who have been exposed to all three vaccines. The sickest people have been those with >25,000. I’m in both of these camps.
So, the question is twofold:
1. Was there a reduction in circulating spike protein.
2. Has the body forgotten about the spike protein.
The twist here is there are more than one spike protein and more than one antibody. Every time a person is exposed to a new variant there are new antibodies for that variant. Each vaccine company used a modified spike protein of their own design, and therefore, the antibodies are different for each.
Per Copilot: “The antibodies your body produces after Pfizer, Moderna, or Janssen vaccination are all directed against the SARS‑CoV‑2 spike protein, but their quantity, durability, and functional profile differ because of the vaccine platform, dose, and immune kinetics.”
So, I’m a bit of a complex case.
What did change for me already:
2/28/2024: Thyroglobulin Antibody = >2,250.0
5/23/2025: Thyroglobulin Antibody = 1698
6/30/2025: Thyroglobulin Antibody = 1543 (9% reduction)
This is a thyroid autoimmune disorder. >2,250.0 is the biggest number two different doctors told me they had ever seen. My body is attacking the thyroid. Following the jammed nicotinic acetylcholine receptor theory, clearing the spike protein from the receptors by using low dose nicotine patches can result in stopping a cascade reaction (described in Dr. Marco Leitzke’s paper) that then results in calming autoimmune reactions. I’m very excited to track this number. I’ve had less problems swallowing since I did the above protocol and I believe it to be related to the low dose nicotine clearing the receptors, though I’m also cleaning up spike protein with Augmented NAC, and not to forget of course I have additionally helped the receptors with Ivermectin.
I am happy to report more as I do more testing.
In the interim, augmentednac.com will be putting their “urine spike test” on their website in the coming months. Dr. Kory’s team I learned recently has been testing the urine test. This is a better way than watching antibodies to show progress. With time the body will forget the spike protein but only after the spike protein is out of the body, which is what the urine test is geared for. Finally, as Dr. Marik mentioned last week Wednesday in a weekly webinar, in Germany they are developing an official way to directly test spike protein. The ability for everyone to be able to test spike protein directly will finally lift the blinders off of the world and we will all be able to see where we are at (even people who are sub-clinical for symptoms). That will be a major step forward for humanity.
-
57 cases of FluMist deaths:<div>Search Results from the VAERS Database
Here’s just a sampling of the write-up’s of a few people who DIED from FluMist:
Write-up: Pt states shortness of breath started 2 weeks ago when he received Flu mist. symptoms mild at that time. he began to have activity limiting symptoms on 19Nov, unable to take a deep breath or sleep because of symptoms. He becomes very SOB with ambulation. Has cough that is only present when he takes a deep breath (nonproductive). Fever and chills over the last 2 weeks, had fever of 102.2 on presentation to emergency room, and satting 91% on room air.
Write-up: On 12/6/10, developed fever & headache. Treated with Advil until 12/9/10; went to PMD; patient vomiting, 12/10/10, vision loss, adm. to E.R. 12/11/10, transferred to PICU. Expired 12/16/10.
Write-up: ADEM secondary to agency from administration of live virus flu vaccine to patient following interferon confusion, fever, coma, death. Contact hospital doctor for details.
Write-up: A serious spontaneous report of INFLUENZA has been received from a non-healthcare professional concerning a male, subsequent to FLUMIST. Neither relevant medical history nor concomitant medications have been reported for this patient. The patient was presumed to be an active employee. On an unknown date, the patient received FLUMIST. Five weeks after receiving FLUMIST, the patient developed symptoms of INFLUENZA. On an unknown date, the patient died. An autopsy was performed. Endemic H1N1 was isolated from the bronchioles. The cause of death was reported as INFLUENZA. The outcome of the event of INFLUENZA was fatal.
Write-up: Member was given the Flumist on 09/22/2011. On 09/25/2011, member was feeling chest pain and not feeling well after dinner and going to the Club. Mamber was found down face in vomit by his friends. CPR was started and 911 activated. Paramedics arrived and continued CPR en route to Hospital where CPR was continued unsuccesfully. Member was pronounced dead at 0215 by Dr.
Write-up: PATIENT PRESENTED WITH VOMITING AND DIARRHEA ON 8 FEB. AAS SHOWED PULM INFILTRATE. PT STARTED ON PO ABX. PT STARTED FEELING FEVERISH ON THE EVENING OF 8 FEB. 9 FEB WOKE UP FEELING LIGHTHEADED AND DIZZY. TAKEN BY AMBULANCE TO ED, WHERE SHE SUFFERED RESP ARREST. CODED 3 TIMES. ADMITTED TO ICU ON VENT. DEVELOPED MULTI-ORGAN SYSTEM FAILURE. DIED ON 10 FEB.
Write-up: Respiratory arrest at home after 4 days of worsening cold symptoms. Driven by family auto to Medical Center and then transfered to PICU at another hospital
Write-up: Patient deceased.
Write-up: A report was received from a physician via a medical representative concerning a 3 year old female. Her medical history, concurrent diseases, and concomitant medications were not reported. On an unknown date, she received nasal FLUMIST. The medical doctor mentioned that two to three years ago a previously healthy 3 year old female died within three days of receiving FLUMIST in her office. The medical representative was uncertain whether symptoms began shortly after dose or three days later. At an unspecified time period after FLUMIST, the patient started vomiting and collapsed. She was taken to hospital three days after receiving FLUMIST and died. The physician stated that all the tests were negative, and the child was afebrile. The medical representative was uncertain whether an autopsy was performed. She also stated that the doctor thought the death was related to FLUMIST. The official cause of death was cardiac arrest. On an unspecified date, the patient died from the event of cardiac arrest, and at the time of reporting the outcome of the event of collapse and vomiting was unknown. According to the reporter the adverse event of cardiac arrest was considered to be serious with the serious criteria of death and the adverse event of vomiting was considered to be non-serious. According to the company physician the adverse event of collapse was considered to be serious with the serious criteria of important medical event. According to the reporter the adverse events were causally related to the suspect drug of FLUMIST.
</div>
-
Not surprisingly, Col. Dan Wattendorf (formerly of DARPA’s ADEPT project – the project that first proposed and started the mRNA approach to address a pandemic) is now working for the Gate’s Foundation Global Health Accelerator: Faster, Smarter Solutions
“Dan Wattendorf
Director, Accelerator
Dan Wattendorf leads the Accelerator program, including its Activator unit, which focuses on exploring, testing and creating new concepts and technologies and activating private-sector entities to advance these new solutions”
